THE PURPOSE OF THIS CORRIGENDUM is to correct errors made inreporting the concentrations of carbon monoxide (CO) causingdilation of pial arteries in three papers published in the AmericanJournal of Physiology-Heart and Circulatory Physiology (1–3).The concentrations of CO in the abscissa of Fig. 2 of Ref. 1and of Figs. 1 and 3 of Ref. 2 should be 0, 10–8, 10–7,10–6, 10–5, 10–6, and 10–5 M insteadof 0, 10–12, 10–11, 10–10, 10–9, 10–8,and 10–7 M, as shown in the published figures; also thehighest concentration of CO in Fig. 3 in Ref. 2 should be 10–4M instead of 10–6 M. Similarly, the concentrations ofCO listed in the abscissa of Figs. 3 and 4 of Ref. 1, Figs.5, 6, and 11 of Ref. 2, and Figs. 1–5 and 7–9 ofRef. 3 should be 0, 10–9, 10–7, and 10–5 Minstead of 0, 10–11, 10–9, and 10–7 M, asshown in the published figures. These errors do not impact onthe conclusions derived from the studies.
REFERENCES
Leffler CW, Nasjletti A, Yu C, Johnson RA, Fedinec AL, Walker N. Carbon monoxide and cerebral vascular tone in newborn pigs. Am J Physiol Heart Circ Physiol 276: H1641–H1646, 1999.[Abstract/Free Full Text]
Leffler CW, Nasjletti A, Johnson RA, Fedinec AL. Contributions of prostacyclin and nitric oxide to carbon monoxide induced cerebrovascular dilation in newborn pigs. Am J Physiol Heart Circ Physiol 280: H1490–H1495, 2001.[Abstract/Free Full Text]
Leffler CW, Fedinec AL, Parfenova H, Jaggar JH. Permissive contributions of NO and prostacyclin in CO-induced cerebrovascular dilation in piglets. Am J Physiol Heart Circ Physiol 289: H432–H438, 2005.[Abstract/Free Full Text]